Here’s a study clearly demonstrating a positive relationship between insecticides and herbicides, but also commonly used solvent chemicals and the development of Parkinson’s syndrome.
Most people are exposed to herbicides and pesticides, not only farmers. Think of your home, school and park lawns, golf courses, driving past garden services at work in the streets and neighborhoods……….. Exposure to these chemicals increases your risk for developing Parkinsons Disease by 33% to 80%.
Testing for chemicals: In practice, it is not currently possible to assess how much chemicals you have in your body other than an exposure history. We do not have tests outside of the research setting. We can however use Autonomic Response Testing to determine which types of chemicals act as stressors on your system. Mercury and other toxic metals are also linked to PD, which can be very easily assessed and safely treated.
Treatment: Depending on the situation and combination of factors involved, there are safe, effective and economical means to decrease the effect of solvents, pesticides and herbicides on the body and to gradually support biochemical pathways that are responsible to clear these chemicals from the body. Tissue repair, cell membrane and dopamine support are also two other components considered.
Currently we do not have studies to guide us on expectations regarding improvement in PD when addressing these individual components, and eagerly await more scientific investigations. W.Vosloo ND MHom
Exposure to pesticides and solvents appears to be a risk factor for Parkinson’s disease, a meta-analysis found.
Based on an analysis of 89 prospective and case-control studies, exposure to bug or weed killers and solvents increased the risk of developing Parkinson’s disease by 33% to 80%, reported Gianni Pezzoli, MD, of the Parkinson Institute, Istituti Clinici di Perfezionamento in Milan, and Emanuele Cereda, MD, PhD, IRCCS University Hospital San Matteo Foundation in Pavia, Italy.
However, the evidence remains “limited, or at least inconclusive,” because of lack of definitive agreement between cohort and case-control studies, the authors cautioned in the May 28 issue of Neurology.
The investigators examined research that considered pesticides, herbicides, insecticides, fungicides, rodenticides, solvents, organochlorines, organophosphates, paraquat, maneb/mancozeb, and DDT, for their relations to Parkinson’s disease.
They also looked at proximity to exposure, such as farming, well-water for drinking, and rural living. The relative risk for Parkinson’s in relation to agricultural jobs was 1.33 (95% CI 1.14-1.56,P<0.001), they stated. Sensitivity analyses based on more than 100 incident cases, they wrote, “confirmed the risk associated with agriculture jobs.”
Paraquat showed a two-fold increase in risk for Parkinson’s, but no association was found to DDT, the authors wrote.
But due to heterogeneity, the results were conflicting. For instance, higher quality studies showed statistical significance for the exposure to solvents, paraquat, and well-water drinking, but they also showed a reduction in risk of Parkinson’s, specifically in relation to insecticides, farming, and well-water drinking. However, high quality studies showed an increase in risk for Parkinson’s for exposure to solvents of more than 30%.
In sensitivity analyses on high-quality studies of more than 200 cases, the risks of Parkinson’s related only to rural living became significant (1.51, 95% CI 1.13-2.03, P=0.006).
The authors offered some explanations for their results. First, heterogeneity in case-control studies seemed to relate mainly to study quality and size, and they had to rely on these data rather than preferred cohort studies to determine etiologic relationships.
In addition, in prospective studies, differences in estimates of exposure to pesticides appeared to depend on the method of ascertainment of Parkinson’s disease, they said.
A study limitation was that the authors did not assess the effect of any secondary causes of Parkinson’s disease in patients recruited from movement disorders clinics.
Nonetheless, “I think the study is actually a big advance in our research knowledge of the relation between chemical exposures and the basic neurological injuries,” said Arch Carson, MD, PhD, of the University of Texas School of Public Health in Houston. “This report is the first to show that there is a positive relationship between not only insecticides and herbicides but also some other solvent chemicals to which many people are exposed and the development of Parkinson’s syndrome.”
But Carson also pointed that the research had clinical limitations. “It does not provide us in medical practice any clear pathway for the treatment of Parkinson’s disease, or even its prevention,” he explained. “We do know there is a significant relationship between various chemical exposures and the development of Parkinson’s disease, as well as a significant relationship with things like family history, smoking, and occupation … but it’s very difficult to put these things into practice except in terms of preventive health, and reducing or eliminating exposures to such suspect materials over time.”
The authors called for more research as did Carson, looking for information on the “specific … and mechanistic relationships between chemical exposure and neurologic injury.”
“We’re only starting to open that black box and understand the details of how that works chemically within the human body, but we’re making progress very fast,” he said. “This study is a benchmark in that process.”
This documentary highlights the revolutionary results of the 10 year, $31 million, NIH study to assess the impact of Chelation Therapy on heart disease and diabetes.
The findings are strongly positive:
Overall improvement 18%
Deaths reduced 7%
Heart Attacks reduced 23%
Strokes reduced 23%
need for Surgery reduced 19%
Hospitalization reduced 23%
Chelation to remove toxic metals from the body is a very important part of treating chronic diseases like chronic fatigue, fibromyalgia, macular degeneration and more – metals interfere with the cells ability to make energy, blood vessels ability to manage blood supply to organs like the heart and brain, which is the start of most chronic and degenerative diseases.
The biggest take home is to understand that some of the most important functions of restorative medicine is to REMOVE bad things from the body, as well as to provide needed nutrients and helpful medicines. This balanced give and take offers more lasting results as you remove roadblocks to healing.
Action item: Insist your doctor does a Urine Toxic Metals test, post provocative, to screen for elevated levels of lead, mercury, aluminum, uranium etc, or call the clinic to ask about our structured testing and treatment protocol for safe sustainable removal of toxic metals as part of treating chronic disease.
Werner Vosloo ND, MHom.
Unleaded Movie Trailer
A documentary exposing the attempt to hide the revolutionary results of the 10 year $31 million NIH study to assess the impact of Chelation Therapy on heart disease and diabetes.
In 1999, cardiologist Roy Heilbron, MD and Angelique Hart, MD began participating in the $31 million double-blind NIH PACT & TACT studies on the effects of Chelation Therapy on heart disease, diabetes, and heart attacks. On Nov. 4, 2012, Dr. Gervasio Lamas, the director of the TACT study presented the stunning results of 10 year study that ivolved over 130 clinics and 1,700 patients at the annual American Heart Association’s 2012 Scientific Sessions. Later that day, Elliott Antman, MD, Director of the AHA, announced that there was not enough information to recommend Chelation Therapy until more studies were done in the future, although TACT was designed to be the definitive study. Normally published the same day as the presentations, the NIH TACT results were not published by the Journal of the American Medicine Association for an unprecedented 5 months, until March 27, 2013, at which time they were ignored and hidden by the mainstream media.
‘Unleaded” takes a look at how heavy metals cause disease, and what Chelation Therapy can do towards possibly curing heart disease, diabetes, and many other diseases by removing heavy metals and toxins from our bodies.
To order the DVD, book a screening in a theater, college or medical university, or to sell DVDs through your holistic clinic, bookshop, or store, please contact us at: info (at) unleadedmovie.com / (505) 695-1319
For more information visit UnleadedMovie.com
3. Mercury, cadmium, inflammation, stress, nutrient deficiencies and infections affect thyroid metabolism adversely, even if your thyroid gland is producing a normal and healthy amount of thyroid hormone.
T4 is the main product of the thyroid gland, with deiodinase enzymes in the liver, kidneys, brain and other organs changing T4 into the metabolically active T3. Most [>95%] PCP’s and endocrinologists do not test for free T3, free T4 and the ratio to reverse T3. Reverse T 3 acts as an anti thyroid, slowing metabolism and contributing to fatigue, pain and disease symptoms.
Action item: Don’t settle for just TSH and fT4 as the only thyroid tests ordered for you. Be pro-active and test your fT3 and reverse T3 to assess all of the important thyroid hormones, which enables your integrated, open minded doctor to adjust your thyroid hormones based on your metabolism and enzyme system function.
Next, test for the toxins known that affect deiodinase enzyme function adversely. We know how to remove these toxins safely, but you will only make this an action item if you test for it.
Effects of low mercury vapour exposure on the thyroid function in chloralkali workers.
Reverse triiodothyronine (rT3) was statistically significantly higher in the mercury exposed subjects, with the free thyroxine (T4)/free T3 ratio also higher in the highest mercury exposed subgroups compared with controls.
Thyroid hormones and methylmercury toxicity.
High exposures to the organometal, methylmercury (MeHg), may perturb neurodevelopmental processes by selectively affecting thyroid hormone homeostasis and function.
Removal of dental amalgam decreases anti-TPO and anti-Tg autoantibodies in patients with autoimmune thyroiditis.
Removal of mercury-containing dental amalgam in patients with mercury hypersensitivity may contribute to successful treatment of autoimmune thyroiditis.
Effects of perinatal exposure to low doses of cadmium or methylmercury on thyroid hormone metabolism.
Thyroid hormone metabolism in fetuses and neonates might be a potential target of Cd and MeHg.
2. Hypothalamic Regulatory Dysfunction or control center dysfunction is a very prevalent factor in thyroid dysfunction seen in fibromyalgia syndrome.
Control center inadequacy in regulating normal release of TSH and basal lowered fT3 levels are a strong cumbersome problem in fibromyalgia. Typically all lab parameters are low normal, with normal levels of TSH and lower levels of fT4 and fT3.
Fibromyalgia patients respond really well to microdoses of T3 therapy clinically. We accompany T3 therapy with cortisol levels to ensure a satisfactory response.
Action item: Get your TSH, fT4, fT3 levels tested to ensure adequate T3 levels to stimulate metabolism and energy production.
Thyroid function in patients with fibromyalgia syndrome.
Patients with fibromyalgia has significantly lower secretion of thyrotropin and thyroid hormones in response to TRH test.
Serum thyroid stimulating hormone in assessment of severity of tissue hypothyroidism.
TSH is a poor measure for estimating the clinical and metabolic severity of primary overt thyroid failure. This is in sharp contrast to the high diagnostic accuracy of TSH measurement for early diagnosis of hypothyroidism.
There is no correlation between the different parameters of target tissues and serum TSH.
Neuroendocrine and hormonal perturbations and relations to the serotonergic system in fibromyalgia patients.
The degree to which TSH can be stimulated is decreased in fibromyalgia patients.
Lowered basal values of free triiodothyronine (fT3) was found in fibromyalgia patients.
This is an outline about the various levels of dysfunction in the thyroid system of patients with chronic disease, especially fibromyalgia, chronic fatigue and post infectious viral, bacterial, mold and lyme related illness. Stay in tune for the two posts to read about a fibromyalgia body has a different thyroid system and therefore requires an individualized thyroid approach.
The reason I share this to explain why “normal” thyroid tests and treatments do not help patients with symptomatic thyroid issues.
1. Thyroiditis is strongly linked to fibromyalgia even if thyroid blood levels are normal.
The studies below demonstrate clearly that Hashimoto’s thyroid autoimmune disease contributes significantly to disease symptoms, regardless of thyroid dysfunction ie even if thyroid hormone levels are apparently normal.
Action item: Ensure you get your antithyroid peroxidase and thyroglobulin antibodies tested.
Hashimoto’s thyroid autoimmune disease responds very well to focused integrated treatment.
Werner Vosloo ND MHom
Symptoms in euthyroid Hashimoto’s thyroiditis: is there a role for autoimmunity itself?
Thyroid auto-immunity per se plays a role in fibromyalgia symptoms and severity, independently from thyroid hormone status.
Rheumatic manifestations of euthyroid, anti-thyroid antibody-positive patients.
46 patients with hashimoto’s thyroid disease had the following findings: 98% joint pain, 59% fibromyalgia, Raynaud’s 28%, ANA auto-immune disease 24% of patients – all with normal thyroid hormone blood levels.
The frequency of thyroid antibodies in fibromyalgia patients and their relationship with symptoms.
FM patients had thyroid autoimmunity similar to the frequency in RA and higher than controls.
Association between thyroid autoimmunity and fibromyalgic disease severity.
Autoimmune thyroiditis is present in an elevated percentage of FM patients, and it has been associated with the presence of typical symptoms of the disease